New PDF release: Acrodermatitis Enteropathica: A Clinician's Guide

By Pooya Khan Mohammad Beigi, Emanual Maverakis

ISBN-10: 3319178180

ISBN-13: 9783319178189

ISBN-10: 3319178199

ISBN-13: 9783319178196

This new textual content offers perception and healing procedures for the coated illness, together with dozens of top of the range pictures, illustrations, and medical tables. themes lined contain the background of the ailment, different types of the sickness, molecular research, epidemiology, results on physique structures, ailment signs, and remedy. Acrodermatitis Enteropathica is an extraordinary affliction of zinc metabolism that happens in a single of 2 types: inborn and bought. If untreated, the ailment is deadly and affected members could die inside of quite a few years, yet when you consider that there's no therapy for the situation, it's serious that physicians are conscious of the symptoms regarding AE and the right remedies on hand for sufferers. A entire consultant to Acrodermatitis Enteropathica is the single booklet of its sort to provide perception into this infrequent disease. Written by means of and for dermatologists, pediatric experts, and ObGYN’s, this e-book comprises one of many greatest scientific learn reports performed on Acrodermatitis Enteropathica over the past decade.​

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Extra resources for Acrodermatitis Enteropathica: A Clinician's Guide

Sample text

When cytoplasmic zinc levels decrease, zinc is released from zincosomes via SLC39A4 transporters. In contrast, high levels of intracellular zinc results in zinc binding with ZIP1 proteins, localized on the zincosomes’ membrane, which prevents the release of vesicular zinc into the cytoplasm. The process by which the ZIP1 molecule prevents the release of zinc is unknown. It is important to note that binding events of the ZIP1 are in binding equilibrium and are not an all-or-none event. Zinc binds to or is released by the ZIP1 depending on its intracellular concentration.

2002;71(1):66–73. 5. Guilbert JJ. The world health report 2002 – reducing risks, promoting healthy life. Educ Health (Abingdon). 2003;16(2):230. 6. Prasad AS. Discovery of human zinc deficiency: its impact on human health and disease. Adv Nutr. 2013;4(2):176–90. 7. Brown KH, et al. International Zinc Nutrition Consultative Group (IZiNCG) technical document #1. Assessment of the risk of zinc deficiency in populations and options for its control. Food Nutr Bull. 2004;25(1 Suppl 2):S99–203. 8. Prasad AS.

53. Dufner-Beattie J, et al. The acrodermatitis enteropathica gene ZIP4 encodes a tissue-specific, zinc-regulated zinc transporter in mice. J Biol Chem. 2003;278(35):33474–81. 54. Dufner-Beattie J, et al. The mouse acrodermatitis enteropathica gene Slc39a4 (Zip4) is essential for early development and heterozygosity causes hypersensitivity to zinc deficiency. Hum Mol Genet. 2007;16(12):1391–9. 55. Geiser J, et al. A mouse model of acrodermatitis enteropathica: loss of intestine zinc transporter ZIP4 (Slc39a4) disrupts the stem cell niche and intestine integrity.

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Acrodermatitis Enteropathica: A Clinician's Guide by Pooya Khan Mohammad Beigi, Emanual Maverakis


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